Study type: In vitro · Status: Verified against declared source
Vasoactive Intestinal Chemical sequence modulates trophoblast-derived cell line function and interaction with phagocytic cells through autocrine pathways.
Scientific reports · 2016
Study scale: The vasoactive intestinal peptide (VIP) is a pleiotropic polypeptide with potent smooth muscle relaxing, vasodilating, pro-secretory and anti-inflammatory effects upon binding high affinity VPAC1 or VPAC2 receptors coupled to stimulatory G protein and adenylate cyclase activation and with lower affinity to PAC1 receptors78.
Abstract only: Open source record
Product or molecular entity relationships
- VIP: Exact entity relationship. Legacy citation custody associates this source with the catalog record; no product-relevance conclusion is implied.
Plain-language verified summary
Question
To transfect Swan 71 cells with a VIP siRNA (Santa Cruz Biotechnology, Dallas, TX, USA), cells were grown at 60% of confluence and arrested for 3 h in Optimem®.
Methods
VIP receptors messenger RNA (mRNA) expression was analyzed by quantitative reverse transcription–polymerase chain reaction (qRT-PCR) as previously described2234.
Scale or participants
The vasoactive intestinal peptide (VIP) is a pleiotropic polypeptide with potent smooth muscle relaxing, vasodilating, pro-secretory and anti-inflammatory effects upon binding high affinity VPAC1 or VPAC2 receptors coupled to stimulatory G protein and adenylate cyclase activation and with lower affinity to PAC1 receptors78.
Key findings
Trophoblast cells migrate and invade the decidual stroma in a tightly regulated process to maintain immune homeostasis at the maternal-placental interface during the first weeks of pregnancy.
Limitations and uncertainty
To investigate whether both VPAC1 and VPAC2 receptor subtypes were involved indistinctively in the modulation of trophoblast cell function, we analyzed cell migration in HTR8 cells over-expressing either VPAC1 or VPAC2 receptors.
Verified against declared source. Verification is limited to the declared source and review scope. It does not mean independent replication or establish efficacy, safety, or suitability.