Study type: In vitro · Status: Evidence verified by machine against declared source
Mitochondrial-derived peptides MOTS-c and humanin attenuate dexamethasone-induced atrophy in human skeletal muscle cells.
Physiological reports · 2026
Study scale: Not published. The text held for this field did not answer this heading, so nothing is shown here and nothing is substituted for it.
Abstract only: Open source record
Product or molecular entity relationships
- Humanin: Exact entity relationship. Legacy citation custody associates this source with the catalog record; no product-relevance conclusion is implied.
- MOTS-c: Catalog source association. Legacy citation custody associates this source with the catalog record; no product-relevance conclusion is implied.
Public plain-language summary
Some fields are not published, because the text held for them did not answer their heading. The published text is limited to the declared source and preserves reported uncertainty. It does not establish efficacy, safety, suitability, or evidence strength.
Research question
Thus, the aim of this study was to explore the possibility of the mitochondrial‐derived peptides HNG and MOTS‐c to mitigate DEXA‐induced atrophy in an in vitro model of primary human skeletal muscle myotubes.
Study design
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Participants or experimental system
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Study scale
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Intervention or exposure
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Comparator
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Outcomes examined
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Key findings
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Limitations and uncertainty
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Product relevance and evidence boundary
This record is a published study, held here with its citation and review status. It is not a statement that any catalog item is effective, safe, or suitable for any use, and nothing in it is a dose or a protocol.
Evidence verified by machine against declared source. Verification is limited to the declared source and review scope. It does not mean independent replication or establish efficacy, safety, or suitability.